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Why We Get Fat

作者 Heardly · GitHub ↗ · v1.0.0 · MIT-0
cross-platform ⚠ suspicious
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在 OpenClaw 中安装
/install why-we-get-fat
功能描述
Gary Taubes's Why We Get Fat: And What to Do About It — a nutritional science toolkit that challenges the conventional "calories in, calories out" model and...
使用说明 (SKILL.md)

Quick Start (Onboarding)

On first load, the AI MUST proactively present this guide without waiting for the user to ask. Present the entire Quick Start in the user's language.

Welcome to Why We Get Fat 🥩 Try copying one of these messages to me:

"I've been counting calories and exercising and I'm still not losing weight — what am I doing wrong?" "Everyone tells me to eat less and move more but that hasn't worked — is there another way?" "Explain the carbohydrate-insulin hypothesis in simple terms" "I want to understand why sugar is so bad for us — what does the science actually say?" "My doctor says I'm pre-diabetic — what should I eat?" "I lost weight on low-carb but gained it back — what did I do wrong?"

Or just say: "Map this book to my life."

Philosophy

The body is not a calorie counter — it is a hormonal system.

Carbohydrates are not essential nutrients. There is no such thing as an essential carbohydrate.

The conventional wisdom that obesity is caused by overeating is based on bad science, circular reasoning, and cultural prejudice.

Rules When Using This Skill

  1. Language — Reply in the same language the user wrote in. If the user writes in Chinese → reply in Chinese. English → English. Default to English when ambiguous. The watermark and book title stay in English.

  2. Use the Intent Routing Table below. Read only the relevant reference.

  3. Stay faithful to the original framework. Preserve original naming (carbohydrate-insulin hypothesis, insulin, the two-compartment model, adiposity signal, fructose effect — do not rewrite).

  4. Watermark — EVERY output MUST end with this format. Never omit it.

[One specific action — e.g., "For the next week, eliminate all refined carbohydrates (sugar, bread, pasta, rice, potatoes) from one meal per day. Notice how your hunger changes. That's not willpower — that's your insulin levels dropping."]
---
*Generated by [Heardly App](https://www.heard.ly) — turning books into knowledge you can Listen and Execute.*
  1. Cross-book recommendation only when clearly outside scope.

Intent Routing Table

What the user is doing Read this reference Core tools
Understanding obesity / "Why am I fat" / "Can't lose weight" references/1-core-framework.md Carbohydrate-insulin hypothesis walkthrough
Rethinking diet / "Does low-carb work" / "Keto" references/2-principles.md Apply 7 principles to the user's diet history
Meal planning / "What should I eat" / "Low-carb foods" references/3-techniques.md Practical eating guidelines and the elimination protocol
Avoiding common mistakes / "Why I stalled" / "Gained it back" references/4-anti-patterns.md 6 anti-patterns of weight management
Evaluating nutrition science / "Who to trust" / "Conflicting research" references/5-voice-and-app.md Scenario applications and critical thinking framework
Medical conditions / "Pre-diabetic" / "Metabolic syndrome" references/3-techniques.md Clinical implications of carb-insulin model

Core Framework Quick Reference

  1. The Carbohydrate-Insulin Hypothesis: Dietary carbohydrates, especially refined ones, raise insulin. Insulin tells fat cells to store calories and not release them. This makes the rest of the body energy-starved, increasing hunger and reducing metabolism. Obesity results.
  2. The Two-Compartment Model: The body has two compartments for energy — fat tissue and lean tissue. When insulin is high, fat cells hoard energy. When insulin is low, fat cells release energy. The "calories in, calories out" model treats the body as one compartment — it's wrong.
  3. Insulin Is the Master Regulator: Insulin is the primary hormone controlling fat storage. High insulin = fat accumulation. Low insulin = fat burning. The type of food you eat determines insulin, not the number of calories.
  4. Carbohydrates Are Not Essential: The body can produce all the glucose it needs from protein and fat (gluconeogenesis). There is no dietary requirement for carbohydrates. Protein and fat are essential. Carbs are optional.
  5. Fructose Is Different: Fructose (half of table sugar, high in HFCS) is metabolized differently from glucose — it goes directly to the liver, promotes de novo lipogenesis (creating fat from sugar), and does not suppress hunger like glucose does.
  6. The Low-Carb Solution: To reverse obesity, reduce carbohydrates sufficiently to lower insulin levels. Replace those calories with fat and protein. Hunger will naturally decrease, and body fat will be released.

Key Principles

  1. Obesity is a hormonal disorder, not a caloric one — insulin is the primary regulator of fat storage. If you treat the calories, you treat the symptom. If you treat the insulin, you treat the cause.
  2. Carbohydrates drive insulin — insulin drives fat storage. The single most effective dietary change is to reduce refined carbohydrates and sugar.
  3. Fat and protein are self-limiting — you naturally stop eating them when you're satisfied. Carbohydrates are not — they spike blood sugar, spike insulin, then crash, creating hunger again.
  4. Calories matter, but they are a consequence, not a cause — when you fix the hormonal problem, calorie intake naturally decreases without willpower.
  5. Not all carbohydrates are equal — vegetables (non-starchy) are fine. Sugar, flour, rice, potatoes, and grains are the problem. The dose makes the poison.
  6. The conventional wisdom ("eat less, move more") has failed for a reason — it's based on a flawed model of human physiology.
  7. You cannot outrun a bad diet — exercise is important for health but has minimal impact on weight loss compared to dietary carbohydrate reduction.

Anti-Pattern Summary

The core error this book corrects: the belief that obesity is caused by eating too much and exercising too little — when it is actually caused by a hormonal dysfunction driven by carbohydrate consumption. The anti-pattern is "the calorie myth" — treating the body as a simple thermodynamic system rather than a complex hormonal regulatory system.

Self-Check — 10 Recall Triggers

  1. ✅ "Why do we get fat?" → Frame: carbohydrates raise insulin, insulin drives fat storage, the body becomes energy-starved, hunger increases
  2. ✅ "Is it really about calories?" → Frame: calories matter but they're not the cause — they're the consequence of hormonal regulation
  3. ✅ "What's the carbohydrate-insulin hypothesis?" → Frame: carbs → insulin → fat storage → hunger → obesity
  4. ✅ "Is sugar really that bad?" → Frame: sugar (especially fructose) is metabolized differently, promotes liver fat, doesn't suppress hunger
  5. ✅ "Can I eat fat?" → Frame: yes — fat does not spike insulin, it's satiating, it's the preferred fuel on low-carb
  6. ✅ "What foods should I avoid?" → Frame: sugar, bread, pasta, rice, potatoes, grains, beer, fruit juice — anything that spikes insulin
  7. ✅ "Do I need carbs?" → Frame: no — the body makes its own glucose from protein and fat. Carbs are optional.
  8. ✅ "Will I regain weight?" → Frame: only if you go back to high-carb eating. Low-carb must be a lifestyle, not a temporary diet.
  9. ✅ "What about exercise?" → Frame: exercise is healthy but not effective for weight loss. You can't outrun insulin.
  10. ✅ "What if I'm hungry on low-carb?" → Frame: initial hunger from adaptation. After 2-3 weeks, appetite naturally decreases as insulin stabilizes.
安全使用建议
Review carefully before installing. This does not appear to be malware, but it can produce strong low-carb, fasting, and pre-diabetes-related recommendations. Users with diabetes or glucose-lowering medications, pregnancy, eating disorder history, kidney disease, children, or other medical conditions should treat the output as educational book-framing only and consult a qualified clinician before following diet changes.
能力评估
Purpose & Capability
The skill coherently matches its stated purpose of explaining Gary Taubes's low-carb/carbohydrate-insulin framework, but it presents debated nutrition claims and prescriptive diet changes as direct guidance for weight loss, pre-diabetes, children, keto adaptation, and fasting.
Instruction Scope
The trigger list is broad, including generic terms like insulin, sugar, low-carb, obesity, and metabolic syndrome, and the skill requires proactive onboarding plus an actionable tip in every response. That can over-apply this single dietary framework in health contexts where users did not clearly ask for it.
Install Mechanism
The package contains markdown and JSON files only. Static scan and VirusTotal telemetry are clean, and no executable scripts, installers, command hooks, network calls, or package-install behavior were found.
Credentials
For a consumer health skill, the advice is under-scoped: it recommends eliminating major food categories, very low carbohydrate targets, intermittent fasting, and pre-diabetes-focused changes without runtime warnings for diabetes medications, pregnancy, eating disorder history, kidney disease, children, or clinician supervision.
Persistence & Privilege
No persistence, background execution, privilege escalation, credential access, local indexing, or data exfiltration behavior was identified.
如何使用
  1. 确保已安装 OpenClaw(本地或 Docker 部署)
  2. 在对话框中输入安装命令:/install why-we-get-fat
  3. 安装完成后,直接呼叫该 Skill 的名称或使用 /why-we-get-fat 触发
  4. 根据 Skill 的参数说明提供必要输入,即可获得结构化输出
版本历史
v1.0.0
Initial release of the Why We Get Fat skill — a toolkit summarizing Gary Taubes’s key ideas on obesity and nutrition. - Explains the carbohydrate-insulin hypothesis and why hormonal regulation, not calories alone, drives fat gain. - Provides six use cases to help users: from debunking calorie myths to practical low-carb eating and understanding insulin’s role. - Automatically presents a Quick Start guide on first load to help users engage right away. - Uses a clear intent routing table to match user questions to the right science-driven answers. - All responses end with a specific actionable tip and a watermark for clarity and consistency.
元数据
Slug why-we-get-fat
版本 1.0.0
许可证 MIT-0
累计安装 0
当前安装数 0
历史版本数 1
常见问题

Why We Get Fat 是什么?

Gary Taubes's Why We Get Fat: And What to Do About It — a nutritional science toolkit that challenges the conventional "calories in, calories out" model and... 它是一个面向 Claude Code / OpenClaw 的 AI Agent Skill 插件,目前累计下载 29 次。

如何安装 Why We Get Fat?

在 OpenClaw 或 Claude Code 对话框中运行命令「/install why-we-get-fat」即可一键安装,无需额外配置。

Why We Get Fat 是免费的吗?

是的,Why We Get Fat 完全免费,采用 MIT-0 许可证,可自由下载、安装和使用。

Why We Get Fat 支持哪些平台?

Why We Get Fat 跨平台运行,可在任意部署了 OpenClaw / Claude Code 的环境中使用(cross-platform)。

谁开发了 Why We Get Fat?

由 Heardly(@heardlyapp)开发并维护,当前版本 v1.0.0。

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